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Welcome to our Technology Offers website
You want access to an innovative offer which is based on the excellence of Inserm, the leading academic research and clinical institute dedicated to human health in Europe? Find out our technologies offers of patents and research tools: here is a sample of our patents.
In vivo studies
Central Nervous System
Ear nose throat disease
Genito Urinary System
The mutant ABC1 allele was generated by homologous recombination, by using a targeting vector containing a neomycin resistance and herpes simplex […]
ADONIS is an agonist-like monoclonal antibody directed against an epitope located on the second extracellular loop of the A2A receptor of adenosine. […]
Expression vectors encoding human 'cellular' fibronectin variants harboring the alternatively spliced Extra Domains B and/or A (or neither) ; […]
Real time constant amperometric detection of NO in tissues/cells.
This gaseous neurotransmitter has a very short half-life (*5 s) and is usually […]
mouse exhibiting a Lox-APL transgene leading to a tissue specific invalidation. […]
The mice are hemizygous for the human apolipoprotein A2 (ApoA2) gene expressed under the control of the homologous promoter (-911/+2045). They are […]
Mice with a cardiac ovexpression of the human beta3-adrenergic receptor were produced by microinjection of a construct with A or C isoform gene under […]
These mouse monoclonal antibodies are directed against human PROS1, an anti-coagulation factor. They are very specific and recognize 70-kDa protein. […]
Rat monoclonal antibody recognizing the CD4 mouse T-cell subset marker. […]
The monoclonal antibody reacts with human and mouse TP53INP1 (both isoforms TP53INP1alpha and […]
This mouse is deficient for TP53INP1 (both isoforms TP53INP1alpha and TP53INP1beta). TP53INP1 is a […]
The monoclonal antibody reacts with human and mouse TP53INP1 (both isoforms TP53INP1alphaƒnand […]
The mutant ABC1 allele was generated by homologous recombination, by using a targeting vector […]
We introduced the Prkcd G510S mutation that we previously associated to a Mendelian cause of SLE in […]
Research tool licensing team
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